Patients with blunt trauma may have their cervical spines cleared clinically if they meet the following criteria:
Not clinically intoxicated (reliable physical exam)
Awake and alert with a GCS of 15
No neck pain or tenderness on exam or motion of neck
No neurological findings (motor or sensory)
No distracting pain
If the above criteria cannot be met, then the C-spine must be cleared with a CT C-spine. If the CT C-spine is negative for fracture, then the C-spine should be clinically reevaluated. If the patient is then clinically sober with a normal GCS and no longer has neck pain or tenderness, the C-spine can be cleared clinically.
All patients with any neurologic symptom consistent with C-spine injury (paresthesia or weakness in the upper extremities) should have a STAT MRI C-spine.
If the patient continues to have neck pain or tenderness AND the patient is clinically sober with a normal GCS AND has no neurologic symptoms consistent with C-spine injury:
The collar may be cleared if an attending radiologist dictates a FINAL negative read on the high quality CT C-spine.
If an attending read will not be completed (e.g., overnight) before the patient is otherwise ready to be discharged, the ligaments can be cleared with a flexion / extension x-ray.
The flexion / extension x-ray must image the area of pain or tenderness and have adequate flexion and extension (15 degrees each direction).
If a patient has persistent midline C-spine pain with a negative CT C-spine +/- negative flex / ex (with adequate visualization of tender area and flexion) then the C-spine is cleared. The patient does not require a Miami J collar / neurosurgery follow-up.
If a flexion / extension x-ray is inadequate or cannot be performed, then an MRI of C-spine is the final method for clearing a C-spine in an awake patient.
If a C-spine is unable to be cleared in an obtunded patient, then a high quality CT C-spine is obtained. If an attending radiologist reads the CT as normal, and there are no focal neurological deficits (symmetric upper and lower extremity strength), the cervical collar may be removed. Any abnormality on the CT C-spine should prompt a neurosurgery consult and a cervical spine MRI.
Patient criteria that warrant BCVI screening:
Cervical bruit in age <50
Expanding cervical hematoma
Focal neurologic deficit
Neuro exam incongruous with head CT findings
Le Fort 2-3 fractures
Bilateral mandible fx with any displacement
Basilar skull fracture (petrous, sphenoid, clivus, occipital condyle)
GCS < 6
C-spine fx with subluxation or ligamentous injury at any level, fracture near vertebral canal
Any C1-3 fracture
Hanging with anoxic injury (any LOC)
Seatbelt sign with swelling, pain, or AMS
Additional specific indicators of high-energy mechanism that warrant BCVI screening:
TBI (GCS < 15) with thoracic injury
Upper rib fractures (1-3)
Scalp degloving
Thoracic vascular injury
CT Head indicated:
• Loss of consciousness (LOC)
• Amnestic to event
• GCS<15
• Patient on anticoagulant or antithrombotic
• Age >65
• Sign of basilar skull fracture
• Concern for skull fracture
▪ If CT head negative AND GCS 15 AND patient not intoxicated AND not on anticoagulant or antithrombotic:
• Discharge home – no need for family observation
• Cermak patients can be returned to Cermak
▪ If CT head negative AND patient is on anticoagulant or antithrombotic: • Discharge home WITH family to watch for 23hrs if INR <3
• If INR >3, keep in OBS for 23hrs
▪ To discharge home, patient must be neurologically intact, not intoxicated, and able to self ambulate. All others should be admitted for 23 hour observation.
▪ Loss of consciousness that is the result of a syncopal episode should get a CT head, an EKG, Troponin, orthostatic blood pressure, and a medicine consult for a
syncope workup (see nonsurgical admit guideline), in addition to the remainder of their symptom-based trauma workup.
BACKGROUND
Blunt chest trauma can lead to a broad range of blunt cardiac injuries (BCI).1 The most common cause of BCI is motor vehicle accidents (50%), pedestrians struck by vehicle (35%), motorcycle crashes (9%) and falls from significant heights (6%).2-4 Diagnosing BCI can be challenging due to polytrauma with multiple injury patterns.
The most common mechanisms to cause BCI are: Direct contusions, indirect trauma, bidirectional trauma (compressing the heart between the sternum and the spine), deceleration injuries, blast injuries, concussive or combined. Direct impact to the chest is considered the most common with subsequent cardiac contusions. It is thought that cardiac injury is most likely to occur when the ventricles are maximally filled at the end of diastole.1 Right ventricular and right atrial injuries are most common whereas left sided heart injury is less common.5 The majority of patients are asymptomatic. Sternal fractures alone are not associated with BCI and do not warrant an echocardiogram in the absence of ECG changes and/or Troponemia.6
Manifestations of BCI can be clinically silent, transient arrhythmias and, rarely, lethal cardiac wall ruptures. A high index of suspicion is required for early diagnosis of BCI. Screening for BCI is performed with an ECG and serologic examination of Troponin. Echocardiography is not a useful screening modality and nuclear medicine scans lack sensitivity and specificity to reliably diagnosis BCI.5 Computed tomography and MRI scans have limited role in the evaluation of BCI.1-6
There are no pathognomonic EKG findings for BCI. The most common arrhythmia is sinus tachycardia.1 However, new onset heart blocks, and ST changes can occur. Previous guidelines stated ECG alone could rule out BCI. However, more recent studies have shown that a subset of patients had BCI and elevated troponin despite an initial normal ECG. For this reason, these guidelines suggest that both a normal ECG and normal Troponin level is required to rule out BCI.6
GUIDELINE ALGORITHM
Screen the following patients for BCI:
Mechanism: Any mechanism with chest wall compression (High speed MVC, pedestrian struck by vehicle, motorcycle accident, Fall from substantial height etc.)
Evidence of blunt chest trauma: Rib fractures, pulmonary contusions, pericardial effusions, cardiac wall motion abnormality on FAST, previously undocumented murmurs, sternal fractures, cardiogenic shock
Concerns on monitor: arrythmias, heart block, ST changes, Physician discretion
SPECIAL CONSIDERATIONS:
Operative Intervention
Patients with BCI are safe to proceed with surgery with the appropriate monitoring
Introduction:
Initial workup should be focused upon accurate anatomic identification and categorization of the aortic injury and assessment of associated injuries. This information will be evaluated jointly by the trauma team and the vascular team to establish treatment priorities and formulate therapeutic plans as outlined below.
Priorities:
Identification of patients with high speed deceleration injuries as warranting CT Scans with IV Contrast of the Chest.
Management of patient’s hemodynamics so as to prevent increased shear stress on the aortic wall and extension of rupture.
Identifying patients who have hypotension and require urgent thoracotomy.
Diagnosis:
Patients with severe blunt chest trauma, especially those with deceleration-type injuries, will be evaluated using CT of the chest with IV contrast.
Blunt aortic injury risk factors:
Accelerating / decelerating injuries > 30MPH, falls >30feet, or sudden compression of chest (i.e., car falling off jack)
Physical exam findings – abrasions or tenderness of chest wall
Abnormalities on CXR
If a patient has an appropriate mechanism (risk factor 1) PLUS one of the other factors – imaging or physical exam findings, then the patient should have a CT of the chest with “arch protocol” to evaluate their aorta.
If the CT arch is abnormal, cardiac / vascular surgery is consulted.
Grading of Injuries:
Injuries will be graded according to the current Society of Vascular Surgery (SVS) guidelines.
Grade 1 – Intimal tear (normal external contour of aorta)
Grade 2 – Intra-mural hematoma
Grade 3 – Pseudoaneurysm
Grade 4 – Pseudoaneurysm with rupture
Initial Medical Management:
All patients with aortic injuries should have active control of blood pressure and heart rate with β blockade to minimize wall shear stress and decrease potential for expansion or rupture.
Therapeutic targets are: SBP < 120, MAP < 80, and pulse < 90.
If SBP >120 or pulse > 90, start Esmolol 500 μg/kg slow bolus over 30 seconds, if BP remains > 120 repeat 500 μg/kg slow bolus over 30 seconds. Use caution, titrate carefully to avoid hypotension (SBP < 80 or MAP < 60)
Start infusion at 50 μg/kg/min
Titrate drip to achieve target SBP, be cautious to avoid hypotension (SBP<80, MAP< 60) especially in patients with potential brain injury
Stable patients without need for ICU admission may receive oral beta-blocker
Treatment:
Grade 1 injuries are generally managed medically with repeat imaging as indicated.
Grade 2 injuries may be managed medically or by TEVAR. Therapeutic plans should be made jointly.
Grade 3 injuries without high-risk features should undergo TEVAR within 24 hours after admission.
Specific therapeutic plans, including order and timing of interventions, should be established jointly after consideration of all associated injuries, most importantly the presence of severe brain injury
Grade 3 injuries with high-risk features should undergo emergency TEVAR.
High risk features include:
Aortic arch hematoma
Ascending aortic, aortic arch, or great vessel involvement
Mediastinal hematoma causing mass effect
Posterior mediastinal hematoma > 10 mm
Lesion to normal aortic diameter ratio > 1.4
Pseudocoarctation of the aorta
Large left hemothorax
Grade 4 injuries should undergo emergency TEVAR. These patients have substantial risk of decompensation and death.
Medical Management:
Anti-platelet therapy:
Given for all grade injuries and for medically and surgically treated patients as allowed by concomitant injuries.
Dose: 81 mg per day.
For patients treated with TEVAR they will take 81 mg for 30 days post-op
For patients treated medically they will take 81 mg until injury resolves on imaging
Blood pressure and heart rate targets:
Non-operative management: transition esmolol drip to oral beta blockers to goal SBP<120 mmHg and pulse <90.
After repair: no blood pressure management required after endovascular coverage of the injury
For patients treated medically, impulse control goals with oral beta blockers will continue until injury resolution seen on imaging
Follow up:
Repeat imaging is not needed inpatient after TEVAR
Repeat imaging inpatient for patients treated medically will be done on select patients as decided by the vascular team
All patients with BTAI should have follow-up CTA scheduled at about 6 weeks post injury
All patients with BTAI should have appropriate follow-up with vascular surgery.
BACKGROUND
Chest trauma occurs in approximately 30-40% of all trauma patients, and roughly one third of these patients will be diagnosed with a pneumothorax, hemothorax, or both. Chest trauma contributes significantly to morbidity and mortality, including half of potentially preventable traumatic deaths.
GUIDELINE ALGORITHM
1. Place chest tube if hemopneumothorax requires evacuation (pneumothorax >35mm or hemothorax >300ml)
a. Pigtail or Large bore chest tube (14-36F) 1-4
Large bore chest tube (28-36F) if chest tube need is emergent.
Pigtail (14F) is effective for PTX and HTX in stable patients
b. One dose of antibiotics to be given just prior to placement to decrease empyema incidence5-6
1st generation cephalosporin
c. Place Pleur-evac to –20 cm H2O wall suction x 24 hours
2. Order post-procedure CXR and daily CXR while tube in place
3. If no radiographic evidence of retained HTX or of PTX enlarging
a. Place tube to water seal
b. CXR after placing on water seal
If PTX worsened, place back to –20 cm H2O wall suction for 48 hours and review with Trauma Fellow or Attending.
If HTX/PTX stable or improved, remove chest tube when output < 100 mL/24hr and no air leak present
4. If radiographic evidence of retained HTX
If within 24 hours, consider placing 2nd chest tube.
Order a non-contrast chest CT on day 3 if HTX is still present on CXR 7
If ≥300mL HTX (formula: V=d2 x l)a present on CT 8-9 then:
VATS on or before day 4 to decrease risk of conversion to thoracotomy, empyema, or need for additional procedures 1, 7-13
Consider intrapleural t-PA 11, 14-18 if poor operative candidate or other indications (t-PA is contraindicated in any patient with intrathoracic arterial bleeding/injury including pulmonary laceration and/or intercostal bleeding). Administration of TPA is not intended to prevent VATS in operative candidates.
TPA/dornase procedure:
Order “Intrapleural Evacuation” powerplanb
Clamp chest tube x 2-4 hours
Roll patient to ensure distribution throughout chest
Unclamp tube and allow drainage
d. Consider IR-guided chest tube if not operative candidate and t-PA not likely to be effective (loculated collection remote from chest tube)
If < 300 mL HTX present, no additional intervention needed 1, 8
Follow chest tube management guidelines per section 3 above
5. If radiographic evidence that PTX is enlarging
a. Review with Trauma Fellow or Attending
b. Place back on wall suction x 48 hours
c. Consider repositioning or replacing chest tube
a d = greatest depth of hemothorax on a single CT image, l = greatest cephalad-caudad length of the hemothorax.
All patients with rib fractures are screened for age, number of rib fractures, presence and laterality of pulmonary contusions, flail chest, and ISS > 15. Patients stratified to highest risk group if they have multiple factors.
Low Risk
Any patient not meeting any factors for medium or high risk
FVC > 2 L
Offered or recommended admission vs discharge to home (attending discretion) for pain control
Medium Risk
≥ 3 rib fractures
Unilateral pulmonary contusion
FVC 1-2 L
Step-down admission
High Risk
Age ≥ 65
≥6 rib fractures
Bilateral pulmonary contusions
Flail segment
ISS > 15
FVC < 1L
ICU admission
All patients receive the following (unless contraindicated):
Acetaminophen 650mg Q6H
Ibuprofen 600mg Q6H
Gabapentin 300mg Q8H
Lidocaine 5% topical Q24H
Consultations to PT/OT/RT
Nursing communication order for: “Please alternate acetaminophen and ibuprofen Q3h”
Nursing communication order for: “Please have incentive spirometer within arm’s reach”
Nursing communication for: “Please document pain score according to unit protocol”
Any patient failing to control pain on the above regimens can be considered for escalation therapy:
Level of care (Floor → Step-Down → ICU)
Titration of gabapentin (300mg → 600mg or 600mg → 900mg) if has been tolerated for the previous 48h without clinical over-sedation
PO Muscle relaxants PRN
PO narcotic PRN
IVP narcotic PRN
PCA narcotic PRN
Lidocaine or ketamine gtt
Rib Fracture Fixation Evaluation
Upon hospital discharge, patient’s OME (oral morphine equivalents) in the previous 24-hours is to be calculated and converted to hydrocodone or oxycodone. Patient is then to be discharged with five-day prescriptions for:
Oxycodone and acetaminophen or hydrocodone-acetaminophen (per OME of last 24h)
Ibuprofen (at last prescribed dose if used in past 24h)
Gabapentin (at last prescribed dose if used in the past 24h)
Lidocaine 5% topical
Patients with blunt trauma to the abdomen should have an evaluation for intra-abdominal injuries. Methods of evaluation are:
Stable Patient
CT abdomen / pelvis
Evaluation for solid organ injury and spine
Unstable Patient
FAST
For use in unstable blunt abdominal patients to identify cavity with hemorrhage
DPL
Useful to identify which cavity is the source of bleeding
Aspirate is most useful in unstable patients
If full lavage is done:
RBC > 100,000 is positive
If patient is stable and refuses imaging, serial abdominal examination can be offered
Must be done frequently and by the same examiner
Only in stable patients
If the patient is getting a CT arch or a CT abdomen / pelvis, the spine can be reconstructed from these images.
A non-contrast CT chest / abdomen with spine reconstruction is less radiation than a dedicated spine CT and may allow for more information such as occult rib fractures or pneumothorax.
Consider non-contrast chest and / or abdomen to reconstruct the spine if unable to clear clinically.
When to consult neurosurgery:
Cervical spine – all fractures
Thoracic spine – any fracture except isolated transverse process
Lumbar spine – any fracture except isolated transverse process